Thyroid Research and Practice

: 2020  |  Volume : 17  |  Issue : 2  |  Page : 62--69

Management of thyroid diseases during pandemic: A narrative review

Mathew John1, Vimal Mavila Veetil2 
1 Providence Endocrine and Diabetes Specialty Centre, Thiruvananthapuram, Kerala, India
2 Department of Endocrinology & Diabetes, Aster MIMS, Kozhikode, Kerala, India

Correspondence Address:
Dr. Mathew John
Providence Endocrine and Diabetes Specialty Centre, Prasanth Nagar, Ulloor, Thiruvananthapuram - 695 011, Kerala

The COVID-19 pandemic caused by the novel coronavirus (SARS-CoV-2) has caused significant morbidity and mortality across the globe. It has resulted in compromised care for patients with chronic diseases. Thyroid diseases are highly prevalent in the world, both in iodine-deficient and -sufficient areas. Viruses can cause thyroid diseases by the direct invasion of the gland, by triggering immune mechanisms, and by the effects of antiviral drugs on the gland. Although there is no signal of a pathogenic mechanism of thyroid diseases by SARS-CoV-2 to date, the high prevalence of thyroid diseases in people affected by this pandemic makes it prudent to study the association between these diseases and to formulate a management plan in the resource-limited setting. In this brief review, we have tried to discuss the relationship between the virus and diseases of the thyroid gland and the management challenges in patients with hypothyroidism, hyperthyroidism, Graves' orbitopathy, and thyroid nodules including cancer. We have also tried to incorporate the advantages and limitations of telemedicine for thyroid disease in the era of a pandemic.

How to cite this article:
John M, Veetil VM. Management of thyroid diseases during pandemic: A narrative review.Thyroid Res Pract 2020;17:62-69

How to cite this URL:
John M, Veetil VM. Management of thyroid diseases during pandemic: A narrative review. Thyroid Res Pract [serial online] 2020 [cited 2022 May 21 ];17:62-69
Available from:;year=2020;volume=17;issue=2;spage=62;epage=69;aulast=John;type=0